Uncommon Descent Serving The Intelligent Design Community

ID Foundations, 14: “Islands” vs “Continents” of complex, specific function — a pivotal issue and debate

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In the current discussion on [Mis-]Representing Natural Selection, UD commenter Bruce David has posed a significant challenge:

A junkers Jumo 004 early Turbojet Engine (Courtesy, Wiki)

. . . it is not obvious that even with intelligence in the picture a major modification of a complex system is possible one small step at a time if there is a requirement that the system continue to function after each such step.

For example, consider a WWII fighter, say the P51 Mustang. Can you imagine any series of incremental changes that would transform it into a jet fighter, say the F80 and have the plane continue to function after each change? To transform a piston engine fighter in to a jet fighter requires multiple simultaneous changes for it to work–an entirely new type of engine, different engine placement, different location of the wings, different cockpit controls and dials, changes to the electrical system, different placement of the fuel tanks, new air intake systems, different materials to withstand the intense heat of the jet exhaust, etc., etc., etc. You can’t make these changes in a series of small steps and have a plane that works after each step, no matter how much intelligence is input into the process.

He then concludes:

Now both a P51 and an F80 are complex devices, but any living organism, from the simplest cell on up to a large multicellular plant or animal, is many orders of magnitude more complex than a fighter plane. If you believe that it is possible to transform a reptile with a bellows lung, solid bones and scales, say, into a bird with a circular flow lung, hollow bones, and feathers by a series of small incremental changes each of which not only results in a functioning organism, but a more “fit” one, then the burden of proof is squarely on your shoulders, because the idea is absurd on the face of it.

In responding, UD Contributor gpuccio clarifies:

consider that engineered modifications can be implemented in a complex organism while retaining the old functionality, and then the new plan can be activated when everything is ready. I am not saying that’s the way it was done, but that it is possible.

For instance, and just to stay simple, one or more new proteins could be implemented using duplicated, non translated genes as origin. Or segments of non coding DNA. That’s, indeed, very much part of some darwinian scenarios.

The difference with an ID scenario is that, once a gene is duplicated and inactivated, it becomes non visible to NS. So, intelligent causes can very well act on it without any problem, while pure randomness, mutations and drift, will be free to operate in neutral form, but will still have the whole wall of probabilistic barriers against them.

[U/d, Dec 30] He goes on to later add:

NS acts as negative selection to keep the already existing information. We see the results of that everywhere in the proteome: the same function is maintained in time and in different species, even if the primary sequence can vary in time because of neutral variation. So, negative NS conserves the existing function, and allow only neutral or quasi neutral variation. In that sense it works againstany emergence of completely new information from the existing one, even if it can tolerate some limites “tweaking” of what already exists (microevolution).

I suppose that darwinists, or at least some of them, are aware of that difficulty as soon as one tries to explain completely new information, such as a new basic protein domain. Not only the darwinian theory cannot explain it, it really works against it.

So, the duplicated gene mechanism is invoked.

The problem is that the duplicated gene, to be free to vary and to leave the original functional island, must be no more translated and no more functional. Indeed, that happens very early in the history of a duplicated gene, because many forma of variation will completely inactivate it as a functional ORF, as we can see all the time with pseudogenes.

So, one of the two:

a) either the duplicated gene remains functional and contributes to the reproduction, so that negative NS can preserve it. In that case, it cannot “move” to new unrelated forms of function.

b) or the duplicated gene immediately becomes non functional, and is free to vary.

The important point is that case a) is completely useless to the darwinian explanation.

Case b) allows free transitions, but they are no more visible to NS, at least not until a new functional ORF (with the necessary regulatory sites) is generated. IOWs, all variation from that point on becomes neutral by definition.

But neutral variation, while free of going anywhere, is indeed free of going anywhere. That means: feedom is accompanied by the huge rising of the probability barriers. As we know, finding a new protein domain by chance alone is exactly what ID has shown to be empirically impossible.

In her attempted rebuttal, contributor Dr Elizabeth Liddle remarks:

I don’t find Behe’s argument that each phylum has a radically different “kernel” very convincing. Sure, prokaryotic cells and eukaryotic cells are different, but, as I said, we have at least one theory (symbiosis) that might explain that. And in any case for non-sexually reproducing organisms, “speciation” is a poor term – what we must postulate is cloning populations that clone along with their symbiotic inclusions. Which is perfectly possible (indeed even we “inherit” parental gut flora).

I think you are making the mistake of assuming that because “phyla” is a term that refers not only to the earliest exemplars of each phylum but also to the entire lineage from each, that those earliest exemplars were as different from each other as we, for example, are from trees, or bacteria. It’s really important to be clear when we are talking longitudinally (adaptation over time) and when laterally (subdivisions of populations into separate lineages).

This was largely in response to Dr V J Torley’s listing of evidence:

What evidence [for the distinctness of main body plans and for abrupt origin of same in the fossil record], Elizabeth? Please have a look here:

http://www.darwinsdilemma.org/pdf/faq.pdf
http://www.darwinsdilemma.org/
http://www.nature.com/news/eni…..ria-1.9714
http://www.arn.org/blogs/index.php/literature

In “The Edge of Evolution”, Dr. Michael Behe argues that phyla were probably separately designed because each phylum has it own kernel that requires design. He also suggests that new orders (or families, or genera – he’s not yet sure which) are characterized by unique cell types, which he thinks must have been intelligently designed, because the number of protein factors in their gene regulatory network (about ten) well exceeds the number that might fall into place naturally (three).

This exchange pivots on the central issue: does complex, multi-part functionality come in easily accessible continents that can be spanned by an incrementally growing and branching tree, or does it normally come in isolated islands in beyond astronomical spaces dominated by seas of non-function, that the atomic level resources of our solar system (our effective universe) or of the observed cosmos as a whole cannot take more than a tiny sample of?

Let’s take the matter in steps of thought:

1 –> Complex, multi-part function depends on having several well-matched, correctly aligned and “wired together” parts that work together to carry out an overall task, i.e. we see apparently purposeful matching and organisation of multiple parts into a whole that carries out what seems to be a goal. The Junkers Jumo 004 Jet engine in the above image is a relevant case in point.

2 –> Ever since Wicken posed the following clip in 1979, this issue of wiring-diagram based complex functional organisation has been on the table as a characteristic feature of life forms that must be properly explained by any successful theory of the causal roots of life. Clip:

‘Organized’ systems are to be carefully distinguished from ‘ordered’ systems.  Neither kind of system is ‘random,’ but whereas ordered systems are generated according to simple algorithms [[i.e. “simple” force laws acting on objects starting from arbitrary and common- place initial conditions] and therefore lack complexity, organized systems must be assembled element by element according to an [[originally . . . ] external ‘wiring diagram’ with a high information content . . . Organization, then, is functional complexity and carries information. It is non-random by design or by selection, rather than by the a priori necessity of crystallographic ‘order.’ [[“The Generation of Complexity in Evolution: A Thermodynamic and Information-Theoretical Discussion,” Journal of Theoretical Biology, 77 (April 1979): p. 353, of pp. 349-65. (Emphases and notes added. Nb: “originally” is added to highlight that for self-replicating systems, the blue print can be built-in.)]

3 –> The question at stake in the thread excerpted from above, is whether there can be an effective, incremental culling-out based on competition for niches and thence reproductive success of sub-populations that will create ever more complex systems that will then appear to have been designed.

4 –> Of course, we must notice that the implication of this claim is that we are dealing with in effect a vast continent of possible functional forms that can be spanned by a gradually branching tree. That’s a big claim, and it needs to be warranted on observational evidence, or it becomes little more than wishful thinking and grand extrapolation in service to an a priori evolutionary materialistic scheme of thought.

5 –> I cases where the function in question has an irreducible core of necessary parts, it is often suggested that something that may have had another purpose may simply find itself duplicated or fall out of use, then fit in with a new use. “Simple.”

6 –> NOT. For, such a proposal faces a cluster of challenges highlighted earlier in this UD series as posed by Angus Menuge [oops!] for the case of the flagellum:

For a working [bacterial] flagellum to be built by exaptation, the five following conditions would all have to be met:

C1: Availability. Among the parts available for recruitment to form the flagellum, there would need to be ones capable of performing the highly specialized tasks of paddle, rotor, and motor, even though all of these items serve some other function or no function.

C2: Synchronization. The availability of these parts would have to be synchronized so that at some point, either individually or in combination, they are all available at the same time.

C3: Localization. The selected parts must all be made available at the same ‘construction site,’ perhaps not simultaneously but certainly at the time they are needed.

C4: Coordination. The parts must be coordinated in just the right way: even if all of the parts of a flagellum are available at the right time, it is clear that the majority of ways of assembling them will be non-functional or irrelevant.

C5: Interface compatibility. The parts must be mutually compatible, that is, ‘well-matched’ and capable of properly ‘interacting’: even if a paddle, rotor, and motor are put together in the right order, they also need to interface correctly.

( Agents Under Fire: Materialism and the Rationality of Science, pgs. 104-105 (Rowman & Littlefield, 2004). HT: ENV.)

8 –> The number of biologically relevant cases where C1 – 5 has been observed: ZERO.

9 –> What is coming out ever more clearly is this:

when a set of matching components must be arranged so they can work together to carry out a task or function, this strongly constrains both the choice of individual parts and how they must be arranged to fit together

A jigsaw puzzle is a good case in point.

So is a car engine — as anyone who has had to hunt down a specific, hard to find part will know.

So are the statements in a computer program — there was once a NASA rocket that veered off course on launch and had to be destroyed by triggering the self-destruct because of — I think it was — a misplaced comma.

The letters and words in this paragraph are like that too.

That’s why (at first, simple level) we can usually quite easily tell the difference between:

A: An orderly, periodic, meaninglessly repetitive sequence: FFFFFFFFFF . . .

B: Aperiodic, evidently random, equally meaningless text: y8ivgdfdihgdftrs . . .

C: Aperiodic, but recognisably meaningfully organised sequences of characters: such as this sequence of letters . . .

In short, to be meaningful or functional, a correct set of core components have to match and must be properly arranged, and while there may be some room to vary, it is not true that just any part popped in in any number of ways can fit in.

As a direct result, in our general experience, and observation, if the functional result is complex enough, the most likely cause is intelligent choice, or design.  

This has a consequence. For, this need for choosing and correctly arranging then hooking up correct, matching parts in a specific pattern implicitly rules out the vast majority of possibilities and leads to the concept of islands of function in a vast sea of possible but meaningless and/or non-functional configurations.

10 –> Consequently, the normal expectation is that complex, multi-part functionality will come in isolated islands. So also, those who wish to assert an “exception” for biological functions like the avian flow-through lung, will need to  empirically warrant their claims. Show us, in short.

11 –> And, to do so will require addressing the difficulty posed by Gould in his last book, in 2002:

. . . long term stasis following geologically abrupt origin of most fossil morphospecies, has always been recognized by professional paleontologists. [The Structure of Evolutionary Theory (2002), p. 752.]

. . . .  The great majority of species do not show any appreciable evolutionary change at all. These species appear in the section [[first occurrence] without obvious ancestors in the underlying beds, are stable once established and disappear higher up without leaving any descendants.” [p. 753.]

. . . . proclamations for the supposed ‘truth’ of gradualism – asserted against every working paleontologist’s knowledge of its rarity – emerged largely from such a restriction of attention to exceedingly rare cases under the false belief that they alone provided a record of evolution at all! The falsification of most ‘textbook classics’ upon restudy only accentuates the fallacy of the ‘case study’ method and its root in prior expectation rather than objective reading of the fossil record. [[p. 773.]

12 –> In that context, the point raised by GP above, that

. . .  once a gene is duplicated and inactivated, it becomes non visible to NS. So, intelligent causes can very well act on it without any problem, while pure randomness, mutations and drift, will be free to operate in neutral form, but will still have the whole wall of probabilistic barriers against them.

. . . takes on multiplied force.

___________

In short, the islands of function issue — rhetorical brush-asides notwithstanding — is real, and it counts.  Let us see how the evolutionary materialism advocates will answer to it. END

PS: I am facing a security headache, so this post was completed on a Linux partition. Linux is looking better than ever, just now. as a main OS . . .

Comments
If you look real hard you might read what I have to say about it elsewhere on this very page. It raises the question - do you people actually read this stuff? I'm not trying to be snarky, but I don't get the impression that you critically analyze anything that suggests support for evolution.ScottAndrews2
January 10, 2012
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An awkward silence ... ;)GCUGreyArea
January 10, 2012
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The process is precisely what is missing.
I just described the process, albeit in a rather sparse form.
One flaw in your reasoning is that, when comparing expectations of the theory to empirical evidence, you consider only the confirming evidence and discard the rest.
Such as ...
The pattern you infer does not include orbital spiderwebs, for example. It actually doesn’t include just about anything at all we see in biology.
???
Take for example whale evolution. It doesn’t fit the pattern at all. It requires a mutation rate quite different from what is observed, in addition to the development of novel features through mutation and selection. So what do you do? You just leave it out because it contradicts your expectations.
WHAT? So whale evolution doesn't fit the pattern we would expect if whales evolved. Do you really believe that all the evidence for whale evolution is ignored by biologists because it contradicts evolution?
What you seem to have trouble understanding is that no one knows whether the differences between genomes is made of mutations and selections. It’s an assumption, a leap. The observation that mutations occur and change the genome does not lead to the conclusion that differences between genomes are made of mutations.
I agree, even though we can see that mutations occur, and we know of multiple sources for possible mutations, and we can observe and even predict their effects, we can never know if any change in a genome is due to a mutation - it could be that god changed it. In the same way we can never know if the existance of a stalactite is due to the deposition of minerals by water, of it god sculpted it. The point is that we have a whole raft of well studied mechanisms by which variation occurs in a genome, we can make good predictions based on this knowledge and no extra entities appear to be required to produce the effects that we see.
Not only does one not logically follow the other, but the conclusion faces enormous hurdles of contradictory evidence. Not least among them is that it forces one to attribute designs that clearly require foresight, planning, and coordination to a process that has neither goals nor the capacity nor the intent to plan for them.
Your reasoning is based on that big assumption - that what you see in nature requires foresight. The problem is that thar the actual research indicates that foresight is not required, and that what we see in nature is more or less what we would expect from the non-foresighted process of evolution.
Given a rodent, generations of mutation and selection give you a bigger or smaller rodent. The process is not consistent with numerous coordinated variations required to add wings, echolocation, and a nervous system that can coordinate them.
More assumptions, driven by a lack of understanding and imagination. Variation can affect many aspects of morphology including limbs and skin folds. The nervous system for hearing already exists, as do ears, and I gather that humans can do primitive echo location with a bit of practice. Have you heard of flying squirrels?
You’ve turned the other way. First evolution explained everything in biology. Now, evolution explains nothing but rather everything in biology supports the pattern of evolution.
??? Are you even reading my comments?
But it doesn’t. By your own words and evidence it supports genetic change over time. It does not support genetic variation and natural selection as the source of that change.
That is a hopelessly confused statement. So the evidence supports the observation of genetic change over time, but variation in the genes is not the source of that variation over time? (BTW Natural selection is not a source of change, it is just a way of saying that different traits in a given environment affect reproduction rates - something that is easy to observe) I think I'll leave it there for the moment.GCUGreyArea
January 10, 2012
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http://blogs.nature.com/news/2012/01/resurrecting-extinct-proteins-shows-how-a-machine-evolves.html I would like to see what those who run this site have to say about the above findings. What is the perspective from an ID point of view. They seem to be strangely silent on this issue?PeterJ
January 10, 2012
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But to characterise every one of the thousands of scientists who have worked on this ‘fluff’ over the last 150 years as stupid, deluded, malicious, deceitful, or pushing a ‘religion’ (the only religion with an extensive mathematical basis and empirical support!) is, I think, wholly unjustified.
What you're saying is not lost on me. At the same time, to reason that what men have believed for 150 years cannot be wrong because that would mean they were deluded or deceived seems off. Have scientists never been wrong? Have there not been plenty of instances in which strongly-held beliefs were held in common by most men of science, and which later turned out to be entirely wrong? There's an extrapolation. If scientists in general can take a position, convincing themselves and others that it is supported by the available evidence when in fact it is not, is it not plausible that they could be likewise deluded in another matter? That's far simpler and more consistent with the body of available evidence than extrapolating from mutations to every genome in every living thing on earth. If someone is willing to accept that extrapolation, why not accept the previous one which is much less of a stretch? Single-celled creatures to pelicans and people via mutation and selection extrapolated from instances of minor phenotypic variation? No problem. Scientists being led to group-think and cling to ideologically motivated conclusions extrapolated from other instances of scientists being led to group-think and clinging to ideologically motivated conclusions? That's a stretch.ScottAndrews2
January 10, 2012
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No, I get it. And I am 100% convinced that you are fascinated! :)ScottAndrews2
January 10, 2012
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It is a list of observations that, when put together form part of a process by which organisms change over time and can generate the patterns observed in the fossil record, in taxonomic classifications, genetics and molecular evolution. It is also a process which, when modeled, produces the same patterns that we see in the fossil record and in genetics.
The process is precisely what is missing. You hardly need a discussion forum to tell someone to go read research. In keeping with the pattern of making assumptions as needed, you assume that I have read nothing. Why? Because surely if I read it I would be convinced. If someone doesn't like kiwi fruit they must not have tried it. One flaw in your reasoning is that, when comparing expectations of the theory to empirical evidence, you consider only the confirming evidence and discard the rest. The available evidence predicts that, starting with any given species, it will vary somewhat in size, shape, and color. This would also be the expectation historically. The pattern you infer does not include orbital spiderwebs, for example. It actually doesn't include just about anything at all we see in biology. And in many cases it doesn't match up at all. Take for example whale evolution. It doesn't fit the pattern at all. It requires a mutation rate quite different from what is observed, in addition to the development of novel features through mutation and selection. So what do you do? You just leave it out because it contradicts your expectations.
That is where genetics comes in, it confirms the pattern and is used to help make predictions that lead to new fossil discoveries.
What you seem to have trouble understanding is that no one knows whether the differences between genomes is made of mutations and selections. It's an assumption, a leap. The observation that mutations occur and change the genome does not lead to the conclusion that differences between genomes are made of mutations. Not only does one not logically follow the other, but the conclusion faces enormous hurdles of contradictory evidence. Not least among them is that it forces one to attribute designs that clearly require foresight, planning, and coordination to a process that has neither goals nor the capacity nor the intent to plan for them. Given a rodent, generations of mutation and selection give you a bigger or smaller rodent. The process is not consistent with numerous coordinated variations required to add wings, echolocation, and a nervous system that can coordinate them. Even an unwarranted extrapolation can slide by, but not when the extrapolated result is a contradiction of the sample. You've turned the other way. First evolution explained everything in biology. Now, evolution explains nothing but rather everything in biology supports the pattern of evolution. But it doesn't. By your own words and evidence it supports genetic change over time. It does not support genetic variation and natural selection as the source of that change.ScottAndrews2
January 10, 2012
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I reasoned on every point and found that it does not lead to the conclusion that diversity in biology is explained by iterations of variation and selection.
That's the bit I missed. The bit where the conclusion is disallowed. I agree that the conclusion is not forced, but I guess what people are looking for is a heuristic to distinguish the bits of diversity that are explained by iterations of variation/selection/drift from those that aren't. We can readily conjure up artificial scenarios whose diversity is fully explained by iterations of variation and selection. Correction, by iterations of mutation-fixation (both selection and drift) IN REPRODUCTIVELY ISOLATED LINEAGES. I know that doesn't change it for you, but it does matter. A simplistic scenario would be a string of meaningless text. Copy it a few times, change the odd letter, delete the copies after an arbitrary 'lifespan', make a few more, recombine strings that are within a few degrees of freedom of sequence identity but not those that are outside it ... carry on for a few million iterations and this simple process will create diversity with all the main characteristics of biological diversity except this elusive quality of 'complex function'. This population will evolve entirely neutrally - there is no 'function', since all points of the space are functional, and there is no selection. And yet I predict that 'species' will emerge - points in the space colonised by multiple 'individuals' clustered together by genetic commonality, and distinguished from other such strings by genetic distance. The recombination rule will create populations exchanging text within, but not between strings that are too 'different'. Now, this isn't biological evolution (though it is evolution). BUT it illustrates the point (or tries to) that diversity is not dependent upon complex function, nor selection. Diversity is about accumulated differences between, and sustained commonality within, multiple lineages subject to imperfect copying. But without retention of the history of the process, I would be at a loss to demonstrate why the process produced the strings it did. "It just did; here's the algorithm ...". So you repeat and it does something different. Now it may be true that complex biological structures or entities exist that cannot be created by selection. I'm not trying to beg any questions with this illustration. But the difference between cats and dogs - that is not a 'complex function'. A cat is complex, a dog is complex, a leopard is complex, a tiger is complex. But the cat-dog distinction does not involve more complexity than the leopard-tiger one, just more (genetic) difference. You seem to accept that the basic processes of evolution can proceed from the common ancestor of leopards and tigers to produce those separate forms. But it cannot progress from a common ancestor of cats and dogs to produce those. Complexity has nothing to do with this asserted circumscription. Of course we cannot answer your challenge to bring forth extinct genes and the selective milieu of extinct organisms in order to demonstrate the principles of evolution at work in a historical setting. No more can I tell you how big was the star in which our elements were fused. You are free, for whatever reasons, to give your designer as big a role in the history of life as you please. But to characterise every one of the thousands of scientists who have worked on this 'fluff' over the last 150 years as stupid, deluded, malicious, deceitful, or pushing a 'religion' (the only religion with an extensive mathematical basis and empirical support!) is, I think, wholly unjustified. You will have to take my word for it, but I argue for evolution because I find it fascinating; I 'get' it, and it provides an excellent fit in my mind between process and the structure of the biological world. I try to convey that fascination, perhaps with little success.Chas D
January 10, 2012
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Yes. But now we’re talking about mutations and genes, not functions. Did you mean to suggest that an accumulation of mutations and other changes results in an accumulation of new functions, features, organs, and behaviors? That would be begging the question, so I’ll assume you didn’t mean that.
yes - that is exactly what is observed - if you did what I suggested and started looking though the thousands of published papers on the subject you might just encounter some of this evidence you are after.
It has not been established that such variations can or do produce increases in function, new organs, features, behaviors, etc.
This is precisely what has been established by researchers - go and look at the research!
This list consists entirely of observations of what was or is and assumptions regarding how it got that way.
It is a list of observations that, when put together form part of a process by which organisms change over time and can generate the patterns observed in the fossil record, in taxonomic classifications, genetics and molecular evolution. It is also a process which, when modeled, produces the same patterns that we see in the fossil record and in genetics.
The fossil record is like a picture of a man in New York and then in Miami and then in L.A. It establishes where he was, not how he got there. You could say that he walked, flew, drove, or was carried and the evidence would support each equally.
That is where genetics comes in, it confirms the pattern and is used to help make predictions that lead to new fossil discoveries. To use your metaphor, genetic evidence suggests walking is the only method of travel, if the man flew he would get there much quicker than would be expected - we don't see these rapid jumps, we see movement consistent with what we know about the rates of change in genomes in different circumstances and for different types of creature.
Now you know that I didn’t just read and dismiss. I reasoned on every point and found that it does not lead to the conclusion that diversity in biology is explained by iterations of variation and selection.
Quite right, you didn't dismiss it, you are utterly failing to understand it whilst throwing in some refuted creationist claims and misconceptions to help demonstrate your lack of knowledge!GCUGreyArea
January 10, 2012
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PeterJ,
Chas D: ‘There is nothing additional separating cats/dogs, as opposed to dogs/wolves, other than time.’ At thet will be why we have so many transitional fossils to look at then.
I don't even know what a transitional fossil is, any more than a 'transitional individual'. Fossils are dead individuals, who were almost certainly closely related to both their parents and (if they had any) their offspring. But they sample discrete points in an assumed continuum of serial inheritance and occasional mutation. That continuum is inferred from the nature of modern individuals and their relationship to their parents and offspring. The sampling of this continuum by discovered fossils is patchy at best, but I am not aware of any fossils that contradict the statement that more time has elapsed since the divergence of cat/dog vs dog/wolf. But I guess one could turn it around. If there is a deep divide between cats and dogs, wouldn't we expect cats and dogs from 30 million year old strata to be as diffferent from each other as are modern cats and dogs? I haven't even looked, but my bet is that those fossils that we have that can be ascribed to 'cat' and 'dog' lineages are less definitively 'different' the further we go back.
Who are you trying to kid?!
Honestly, sincerely? No-one.Chas D
January 10, 2012
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ScottAndrews2, As you appear to be playing the advocate I'll join in!
Nearly every cited example involves loss of function.
How "nearly" is nearly? It seems to me that one thing can be broken in many different ways. "Never" would be different. So, specifically, what does the ratio of "gain of function" mutations to "loss of function" mutations have to be to make what is being discussed impossible? 1:100? 1:10000000?
This borders on question-begging.
You just admitted that "nearly" every example was loss of function, therefore there were >0 gain of function. Indulge me. Get specific! Give me an example of such a "gain of function" that you are aware of! Give me 5!
First you’re assuming that genetic similarities mean common ancestry.
Common ancestry is accepted by Behe. It's well supported. It's not an assumption.
Not all things that share common elements have common ancestors.
Fer'instance?
It has not been established that such variations can or do produce increases in function, new organs, features, behaviors, etc.
No, not to your satisfaction. But nonetheless as a working assumption it seems to be usable and productive.
The fossil record is like a picture of a man in New York and then in Miami and then in L.A. It establishes where he was, not how he got there. You could say that he walked, flew, drove, or was carried and the evidence would support each equally.
What is your opinion of the evolution of mammalian auditory ossicles? Does hearing count as a gain of function?Peter Griffin
January 10, 2012
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Yeah mutations accumulate but the question is how was it determined mutations are random in any sense of the word? As for nested hierarchies, based on what, exactly? Nested hierarchy patterns are man-made constructs...Joe
January 10, 2012
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Understood. Geneticist Giuseppe Semonti touched on this in "Why is a Fly Not a Horse?"- his main point is all we know about cats and dogs is that a cat is born when there is a successful mating of two cats (and a dog is born when there is a successful mating of two dogs)- gens control traits and neither "dog" nor "cat" (nor "human") is a trait:
”The scientist enjoys a privilege denied the theologian. To any question, even one central to his theories, he may reply “I’m sorry but I do not know.” This is the only honest answer to the question posed by the title of this chapter. We are fully aware of what makes a flower red rather than white, what it is that prevents a dwarf from growing taller, or what goes wrong in a paraplegic or a thalassemic. But the mystery of species eludes us, and we have made no progress beyond what we already have long known, namely, that a kitty is born because its mother was a she-cat that mated with a tom, and that a fly emerges as a fly larva from a fly egg.”
Meaning genes influence development bt do not determine it. Also a targeted search is not unguided evolution. The barrier unguided evolution can't cross? More than two new protein-to-protein binding sites, which means you are getting to start with what needs to be explained and still can't get much from there.Joe
January 10, 2012
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And what predictions can be made of accumulations of random mutations? Please be specific.
On its own, nothing. It is an observation - mutations accumulate. One pattern that can be predicted when you add differential reproduction rates is that only near neutral or beneficial mutations will accumulate, and the resulting generations will produce a pattern of nested hierarchies.GCUGreyArea
January 10, 2012
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GCU, Just for the sheer heck of it, let's go through this.
Offspring inherit a combination of parents genes.
Yes.
Offspring can also have mutations no present in their genome.
Yes.
Different combinations of genes produce different individuals.
Yes.
Mutations can alter the way cells function and the way an organism appears and operates.
Yes, although the extent of this is unclear. Nearly every cited example involves loss of function. This borders on question-begging.
Organisms with certain traits may be more or less likely to reproduce well in a given environment.
Yes.
A slight variation in a particular trait can give an advantage or disadvantage, resulting in more or less offspring.
Yes. What other kind of variation is there?
Mutations happen at a certain rate.
Yes.
Mutations and other changes accumulate.
Yes. But now we're talking about mutations and genes, not functions. Did you mean to suggest that an accumulation of mutations and other changes results in an accumulation of new functions, features, organs, and behaviors? That would be begging the question, so I'll assume you didn't mean that.
Different species will sometimes share similar genes.
Yes. This is only significant if we start with certain assumptions as explained above.
The rate of accumulation and the genetic similarities can be used to estimate when two species shared a common ancestor.
See the above comment. First you're assuming that genetic similarities mean common ancestry. Not all things that share common elements have common ancestors. That certain things reproduce and have ancestors does not change that. Second, this assumes that the variation between species is the result of such accumulated genetic variations. It has not been established that such variations can or do produce increases in function, new organs, features, behaviors, etc. So again, this is begging the question, rephrasing the assumption.
Examples of these ancestors in the fossil record correlate with this.
First, that they are ancestors at all is more evident in some cases than in others. Second, as stated, that the difference between one specimen and the next is the result of variation and selection has not been established. Is there so much as a single instance in which a genetic variation and the selection of it can be determined from fossils? The fossil record is like a picture of a man in New York and then in Miami and then in L.A. It establishes where he was, not how he got there. You could say that he walked, flew, drove, or was carried and the evidence would support each equally.
Taxonomic classifications also correlate with the above.
See the above. This list consists entirely of observations of what was or is and assumptions regarding how it got that way. Combining relevant facts into a list to make a point can be powerful. It often works even when the facts don't particularly add up to anything. Some people, when presented with a big list of stuff, much of which is plainly true, are easily persuaded that one follows from or supports the other. Salesmen know that if you want someone to buy something you start by asking them a bunch of questions to which they will answer "yes." Now you know that I didn't just read and dismiss. I reasoned on every point and found that it does not lead to the conclusion that diversity in biology is explained by iterations of variation and selection. Since none of this leads to that conclusion, what does? Please, let no one go back to "we sent you lots of stuff and you ignored it." I did not, and you do not get credit for quantity of bullet points or just for bothering to make a list at all.ScottAndrews2
January 10, 2012
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Well I can take the alleged evidence for universal common descent and use it to support a common design.
Well, to be clear, I was not talking about universal common descent. Somewhere in between universal descent and you being identifiably the child of your parents, there is a barrier being asserted that unguided evolution cannot cross. I am trying to ascertain where people think that barrier is actually located, within a clade of anyone's choosing. KF explicitly talks of families - cat vs dog as the example. Thus unguided evolution is admitted to account for genetic differences between any two cat types (eg leopard and tiger) but not between the various cat species and the various dog species. Likewise, the genetic commonality between cat species in this 'unguided' clade can only be due to common descent, however it first originated. Under 'unguided' evolution, populations change due to the accumulation of changes in lineages of descent, and diverge due to branching caused by long-term isolation. Therefore, the retained similarities between such populations are due to common descent - their common genes were obtained from their common ancestors, however those ancestors got 'em. So common descent is a valid inference in such a scenario. The differences between the populations are all due to evolution, while their similarities are due to common descent. But the same thing happens when we look at the dog/cat split. There are similarities that are indistinguishable from the correspondence that is agreed to show common descent within the cats. The only difference is that there are fewer of them, and more differences. So the question is really how one decides that common descent is a valid cause of the same genes being observed in different cats, but is not valid when one observes the same genes in cats and dogs.
And no, that does not mean all organisms were designed as they are today. That means all organisms of today are descended from the originals.
Well, yeah. From the point at which the designer stopped designing, all differences can only be due to unguided evolution. But if (when we compare leopards and tigers, say) we see similarities that are due to common descent and differences that are due to evolutionary divergence, what are we to honestly conclude when we encounter that same mix of similarities and differences between cats and dogs, differing only in the proportions ascribed to 'similar' and 'different?Chas D
January 10, 2012
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Scott, It's comical. You're asking for evidence for evolution, and over and over again you're given it. Yet you are unwilling to listen. You remind me of Steve Martin in "The man with two brains", asking his recently deceased wife for a sign from heaven if she dissaproves of him starting a new relationship... http://www.youtube.com/watch?v=lmivsA3iJw8lastyearon
January 10, 2012
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Champignon, So you thought that when I referred to the 'cornerstone of biology' and to 'genetic variations and the selection of them; that I was talking about two different things. I thought I was pretty clear. I realize that there's more to evolution than variation and selection. But when we're talking about the origin of species or anything new, it's about precisely that. Note to self: Be even more deliberately careful to use the exact same words over and over in boring repetition for fear that I'll be accused of asking different questions. You haven't supplied it. You offered a laundry list of things other than evolution by variation and selection. Some of them even explicitly had nothing to do with selection. Geographical separation, for example. It is not selection when a gull chooses to breed with nearby gulls rather than gulls 100 miles away. And if it was it would still be a poor example because if you put the two together they would breed anyway. I could try rephrasing. You clearly believe that there was once a wingless creature, and many generations later as a result of variation and selection, there were winged creatures that flew. (That's an abbreviated version. I'm sure there's more to it.) I'm sure you also believe that before geckos had feet enabling them to cling to walls, they had an ancestor that did not. You believe that before there were spiders that weaved orbital webs there were spiders that did not, and before them spiders that produced no type of silk at all. And in each case the difference was genetic changes over many generations, some of which were selected. Correct me if I'm wrong. I'm just summarizing. The question is, why do you think that? I'm not asking you to explain any one of these things specifically. (That's me being reasonable again.) But what is explained by variation and natural selection so that you (and I) can reasonably infer that it explains such things also? You must realize that the only meaningful answer to that question is an explanation of something using variation and natural selection. Inference and extrapolation are fine, but they do require a basis. What is it?ScottAndrews2
January 10, 2012
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Scott's locutions become increasingly awkward as he moves the goalposts. Before he was asking for examples of “how the cornerstone of biology explains something in biology." Unfortunately for Scott, we supplied what he requested. That put him in an awkward spot, so he had to deny that the question had been answered and change it. Now he'd like us to "specify genetic variations and the selection of such variations", presumably identifying the base pairs involved.champignon
January 10, 2012
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Wrong again, as usual. GCU:
In what way
Newton’s First Rule applies, as does parsimony and Occam, which means the explanatory filter applies. And all of that tells us that if necessity/ law and/ or random events can explain it then we do not add unnecessary entities. Therefor not everything is consistent with design. So again I ask you: And what predictions can be made of accumulations of random mutations? Please be specific. Or you can continue to hide behind your strawman.Joe
January 10, 2012
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Me:
Everything is consistent with ID – unless you know something about the designer that places constraints on their actions.
Joe:
Wrong again, as usual.
In what way - that somethings cannot be created by design, or that we know some things about the hypothesized designer that constrain their actions?GCUGreyArea
January 10, 2012
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It consists of items that do not specify genetic variations or the selection of such variations.
What does that sentence even mean?GCUGreyArea
January 10, 2012
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I did address it when you first posted it. It consists of items that do not specify genetic variations or the selection of such variations. That makes them poor evidence to support a process consisting of genetic variations and the natural selection of them. I'm just noting that when reminding me later of your list, you don't single anything out. You just lump it all together. That suggests that either nothing on the list stands out to you or that you think that the quantity of items itself makes it more substantial - the "mountains of evidence" argument.ScottAndrews2
January 10, 2012
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Scott,
That Champignon refers to “a list” rather than any specific item demonstrates that even he is unwilling to take the risk of citing a specific example.
I didn't refer to "a list", I referred to "this list" and then provided it. You seem to be afraid of addressing it. Why?champignon
January 10, 2012
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Wrong again, as usual. Newton's First Rule applies, as does parsimony and Occam, which means the explanatory filter applies. And all of that tells us that if necessity/ law and/ or random events can explain it then we do not add unnecessary entities. So again I ask you: And what predictions can be made of accumulations of random mutations? Please be specific. Or you can continue to hide behind your strawman.Joe
January 10, 2012
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Champignon, I'm not responding at random. If the response is trivial I'll say so and why. If someone attempts to distract by trifling over words I'll point that out. I mention how reasonable I to remind onlookers that I'm asking a very, very simple question. The boredom - okay, I can keep that to myself. If you don't like me stating that the answers are trivial, provide less trivial answers or argue why the answers you gave are not trivial. If you don't want me to point out when you're trifling, don't trifle. Or argue why it's not trifling. Who said that supported extrapolation is a cardinal sin? No one, so why say it? You're telling me that you're unhappy with my responses. That's okay. You don't have to be happy with them. But why do you think that summarizing them (to the extent that you do so accurately) is a useful criticism of them? If you don't like the output then change the input.ScottAndrews2
January 10, 2012
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Everything is consistent with ID - unless you know something about the designer that places constraints on their actions.GCUGreyArea
January 10, 2012
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#2 should read, "Ignore your opponent's answer, or claim that it's trivial and demand a real answer."champignon
January 10, 2012
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Here's Scott's algorithm: 1. Ask a question. 2. Ignore your opponent's answer, or else claim that it's trivial and demand 3. Restate your question as if it hadn't been answered. 4. Comment on how reasonable you're being. 5. Occasionally complain that you're being "trifled with." 6. Pretend that justified extrapolation is a cardinal sin. 7. Talk about how bored you are. 8. Repeat.champignon
January 10, 2012
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All of that is in line with baraminology and ID. And what predictions can be made of accumulations of random mutations? Please be specific.Joe
January 10, 2012
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