From Mike Klymkowsky at PLOS:
Through his studies on peas, Gregor Mendel was the first to clearly identify some of the rules for the behavior of these inheritable factors using highly stereotyped, and essentially discontinuous traits – a pea was either yellow or green, wrinkled or smooth. Such traits, while they exist in other organisms, are in fact rare – an example of how the scientific exploration of exceptional situations can help understand general processes, but the downside is the promulgation of the idea that genes and traits are somehow discontinuous – that a trait is yes/no, displayed by an organism or not – in contrast to the realities that the link between the two is complex, a reality rarely directly addressed (apparently) in most introductory genetics courses. Understanding such processes is critical to appreciating the fact that genetics is often not destiny, but rather alterations in probabilities (see Cooper et al., 2013). Without such an more nuanced and realistic understanding, it can be difficult to make sense of genetic information. More.
Can’t speak for others but here at Uncommon Descent, we have been thinking that for quite some time. We’re not shocked but we are annoyed when discredited textbook Darwinism is treated as the norm and the true state of affairs is treated as a big surprise.
See also: Bale monkeys more closely related to sister species than same species in different locationsThe “biological species concept” is yet another textbook dead zone.
Girl got mostly a double set of her dad’s genes, is almost a twin. She has some problems but she is 11 years old. Yes, that was the sound of another lectern splintering in the near distance.
Almost one in five genes’ coding status is unresolved Researchers: We believe that the three reference databases currently overestimate the number of human coding genes by at least 2000, complicating and adding noise to large-scale biomedical experiments.
Do all genes affect every complex trait? Veronique Greenwood: The roots of many traits, from how tall you are to your susceptibility to schizophrenia, are far more tangled. In fact, they may be so complex that almost the entire genome may be involved in some way