Uncommon Descent Serving The Intelligent Design Community

ID Foundations, 14: “Islands” vs “Continents” of complex, specific function — a pivotal issue and debate

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In the current discussion on [Mis-]Representing Natural Selection, UD commenter Bruce David has posed a significant challenge:

A junkers Jumo 004 early Turbojet Engine (Courtesy, Wiki)

. . . it is not obvious that even with intelligence in the picture a major modification of a complex system is possible one small step at a time if there is a requirement that the system continue to function after each such step.

For example, consider a WWII fighter, say the P51 Mustang. Can you imagine any series of incremental changes that would transform it into a jet fighter, say the F80 and have the plane continue to function after each change? To transform a piston engine fighter in to a jet fighter requires multiple simultaneous changes for it to work–an entirely new type of engine, different engine placement, different location of the wings, different cockpit controls and dials, changes to the electrical system, different placement of the fuel tanks, new air intake systems, different materials to withstand the intense heat of the jet exhaust, etc., etc., etc. You can’t make these changes in a series of small steps and have a plane that works after each step, no matter how much intelligence is input into the process.

He then concludes:

Now both a P51 and an F80 are complex devices, but any living organism, from the simplest cell on up to a large multicellular plant or animal, is many orders of magnitude more complex than a fighter plane. If you believe that it is possible to transform a reptile with a bellows lung, solid bones and scales, say, into a bird with a circular flow lung, hollow bones, and feathers by a series of small incremental changes each of which not only results in a functioning organism, but a more “fit” one, then the burden of proof is squarely on your shoulders, because the idea is absurd on the face of it.

In responding, UD Contributor gpuccio clarifies:

consider that engineered modifications can be implemented in a complex organism while retaining the old functionality, and then the new plan can be activated when everything is ready. I am not saying that’s the way it was done, but that it is possible.

For instance, and just to stay simple, one or more new proteins could be implemented using duplicated, non translated genes as origin. Or segments of non coding DNA. That’s, indeed, very much part of some darwinian scenarios.

The difference with an ID scenario is that, once a gene is duplicated and inactivated, it becomes non visible to NS. So, intelligent causes can very well act on it without any problem, while pure randomness, mutations and drift, will be free to operate in neutral form, but will still have the whole wall of probabilistic barriers against them.

[U/d, Dec 30] He goes on to later add:

NS acts as negative selection to keep the already existing information. We see the results of that everywhere in the proteome: the same function is maintained in time and in different species, even if the primary sequence can vary in time because of neutral variation. So, negative NS conserves the existing function, and allow only neutral or quasi neutral variation. In that sense it works againstany emergence of completely new information from the existing one, even if it can tolerate some limites “tweaking” of what already exists (microevolution).

I suppose that darwinists, or at least some of them, are aware of that difficulty as soon as one tries to explain completely new information, such as a new basic protein domain. Not only the darwinian theory cannot explain it, it really works against it.

So, the duplicated gene mechanism is invoked.

The problem is that the duplicated gene, to be free to vary and to leave the original functional island, must be no more translated and no more functional. Indeed, that happens very early in the history of a duplicated gene, because many forma of variation will completely inactivate it as a functional ORF, as we can see all the time with pseudogenes.

So, one of the two:

a) either the duplicated gene remains functional and contributes to the reproduction, so that negative NS can preserve it. In that case, it cannot “move” to new unrelated forms of function.

b) or the duplicated gene immediately becomes non functional, and is free to vary.

The important point is that case a) is completely useless to the darwinian explanation.

Case b) allows free transitions, but they are no more visible to NS, at least not until a new functional ORF (with the necessary regulatory sites) is generated. IOWs, all variation from that point on becomes neutral by definition.

But neutral variation, while free of going anywhere, is indeed free of going anywhere. That means: feedom is accompanied by the huge rising of the probability barriers. As we know, finding a new protein domain by chance alone is exactly what ID has shown to be empirically impossible.

In her attempted rebuttal, contributor Dr Elizabeth Liddle remarks:

I don’t find Behe’s argument that each phylum has a radically different “kernel” very convincing. Sure, prokaryotic cells and eukaryotic cells are different, but, as I said, we have at least one theory (symbiosis) that might explain that. And in any case for non-sexually reproducing organisms, “speciation” is a poor term – what we must postulate is cloning populations that clone along with their symbiotic inclusions. Which is perfectly possible (indeed even we “inherit” parental gut flora).

I think you are making the mistake of assuming that because “phyla” is a term that refers not only to the earliest exemplars of each phylum but also to the entire lineage from each, that those earliest exemplars were as different from each other as we, for example, are from trees, or bacteria. It’s really important to be clear when we are talking longitudinally (adaptation over time) and when laterally (subdivisions of populations into separate lineages).

This was largely in response to Dr V J Torley’s listing of evidence:

What evidence [for the distinctness of main body plans and for abrupt origin of same in the fossil record], Elizabeth? Please have a look here:

http://www.darwinsdilemma.org/pdf/faq.pdf
http://www.darwinsdilemma.org/
http://www.nature.com/news/eni…..ria-1.9714
http://www.arn.org/blogs/index.php/literature

In “The Edge of Evolution”, Dr. Michael Behe argues that phyla were probably separately designed because each phylum has it own kernel that requires design. He also suggests that new orders (or families, or genera – he’s not yet sure which) are characterized by unique cell types, which he thinks must have been intelligently designed, because the number of protein factors in their gene regulatory network (about ten) well exceeds the number that might fall into place naturally (three).

This exchange pivots on the central issue: does complex, multi-part functionality come in easily accessible continents that can be spanned by an incrementally growing and branching tree, or does it normally come in isolated islands in beyond astronomical spaces dominated by seas of non-function, that the atomic level resources of our solar system (our effective universe) or of the observed cosmos as a whole cannot take more than a tiny sample of?

Let’s take the matter in steps of thought:

1 –> Complex, multi-part function depends on having several well-matched, correctly aligned and “wired together” parts that work together to carry out an overall task, i.e. we see apparently purposeful matching and organisation of multiple parts into a whole that carries out what seems to be a goal. The Junkers Jumo 004 Jet engine in the above image is a relevant case in point.

2 –> Ever since Wicken posed the following clip in 1979, this issue of wiring-diagram based complex functional organisation has been on the table as a characteristic feature of life forms that must be properly explained by any successful theory of the causal roots of life. Clip:

‘Organized’ systems are to be carefully distinguished from ‘ordered’ systems.  Neither kind of system is ‘random,’ but whereas ordered systems are generated according to simple algorithms [[i.e. “simple” force laws acting on objects starting from arbitrary and common- place initial conditions] and therefore lack complexity, organized systems must be assembled element by element according to an [[originally . . . ] external ‘wiring diagram’ with a high information content . . . Organization, then, is functional complexity and carries information. It is non-random by design or by selection, rather than by the a priori necessity of crystallographic ‘order.’ [[“The Generation of Complexity in Evolution: A Thermodynamic and Information-Theoretical Discussion,” Journal of Theoretical Biology, 77 (April 1979): p. 353, of pp. 349-65. (Emphases and notes added. Nb: “originally” is added to highlight that for self-replicating systems, the blue print can be built-in.)]

3 –> The question at stake in the thread excerpted from above, is whether there can be an effective, incremental culling-out based on competition for niches and thence reproductive success of sub-populations that will create ever more complex systems that will then appear to have been designed.

4 –> Of course, we must notice that the implication of this claim is that we are dealing with in effect a vast continent of possible functional forms that can be spanned by a gradually branching tree. That’s a big claim, and it needs to be warranted on observational evidence, or it becomes little more than wishful thinking and grand extrapolation in service to an a priori evolutionary materialistic scheme of thought.

5 –> I cases where the function in question has an irreducible core of necessary parts, it is often suggested that something that may have had another purpose may simply find itself duplicated or fall out of use, then fit in with a new use. “Simple.”

6 –> NOT. For, such a proposal faces a cluster of challenges highlighted earlier in this UD series as posed by Angus Menuge [oops!] for the case of the flagellum:

For a working [bacterial] flagellum to be built by exaptation, the five following conditions would all have to be met:

C1: Availability. Among the parts available for recruitment to form the flagellum, there would need to be ones capable of performing the highly specialized tasks of paddle, rotor, and motor, even though all of these items serve some other function or no function.

C2: Synchronization. The availability of these parts would have to be synchronized so that at some point, either individually or in combination, they are all available at the same time.

C3: Localization. The selected parts must all be made available at the same ‘construction site,’ perhaps not simultaneously but certainly at the time they are needed.

C4: Coordination. The parts must be coordinated in just the right way: even if all of the parts of a flagellum are available at the right time, it is clear that the majority of ways of assembling them will be non-functional or irrelevant.

C5: Interface compatibility. The parts must be mutually compatible, that is, ‘well-matched’ and capable of properly ‘interacting’: even if a paddle, rotor, and motor are put together in the right order, they also need to interface correctly.

( Agents Under Fire: Materialism and the Rationality of Science, pgs. 104-105 (Rowman & Littlefield, 2004). HT: ENV.)

8 –> The number of biologically relevant cases where C1 – 5 has been observed: ZERO.

9 –> What is coming out ever more clearly is this:

when a set of matching components must be arranged so they can work together to carry out a task or function, this strongly constrains both the choice of individual parts and how they must be arranged to fit together

A jigsaw puzzle is a good case in point.

So is a car engine — as anyone who has had to hunt down a specific, hard to find part will know.

So are the statements in a computer program — there was once a NASA rocket that veered off course on launch and had to be destroyed by triggering the self-destruct because of — I think it was — a misplaced comma.

The letters and words in this paragraph are like that too.

That’s why (at first, simple level) we can usually quite easily tell the difference between:

A: An orderly, periodic, meaninglessly repetitive sequence: FFFFFFFFFF . . .

B: Aperiodic, evidently random, equally meaningless text: y8ivgdfdihgdftrs . . .

C: Aperiodic, but recognisably meaningfully organised sequences of characters: such as this sequence of letters . . .

In short, to be meaningful or functional, a correct set of core components have to match and must be properly arranged, and while there may be some room to vary, it is not true that just any part popped in in any number of ways can fit in.

As a direct result, in our general experience, and observation, if the functional result is complex enough, the most likely cause is intelligent choice, or design.  

This has a consequence. For, this need for choosing and correctly arranging then hooking up correct, matching parts in a specific pattern implicitly rules out the vast majority of possibilities and leads to the concept of islands of function in a vast sea of possible but meaningless and/or non-functional configurations.

10 –> Consequently, the normal expectation is that complex, multi-part functionality will come in isolated islands. So also, those who wish to assert an “exception” for biological functions like the avian flow-through lung, will need to  empirically warrant their claims. Show us, in short.

11 –> And, to do so will require addressing the difficulty posed by Gould in his last book, in 2002:

. . . long term stasis following geologically abrupt origin of most fossil morphospecies, has always been recognized by professional paleontologists. [The Structure of Evolutionary Theory (2002), p. 752.]

. . . .  The great majority of species do not show any appreciable evolutionary change at all. These species appear in the section [[first occurrence] without obvious ancestors in the underlying beds, are stable once established and disappear higher up without leaving any descendants.” [p. 753.]

. . . . proclamations for the supposed ‘truth’ of gradualism – asserted against every working paleontologist’s knowledge of its rarity – emerged largely from such a restriction of attention to exceedingly rare cases under the false belief that they alone provided a record of evolution at all! The falsification of most ‘textbook classics’ upon restudy only accentuates the fallacy of the ‘case study’ method and its root in prior expectation rather than objective reading of the fossil record. [[p. 773.]

12 –> In that context, the point raised by GP above, that

. . .  once a gene is duplicated and inactivated, it becomes non visible to NS. So, intelligent causes can very well act on it without any problem, while pure randomness, mutations and drift, will be free to operate in neutral form, but will still have the whole wall of probabilistic barriers against them.

. . . takes on multiplied force.

___________

In short, the islands of function issue — rhetorical brush-asides notwithstanding — is real, and it counts.  Let us see how the evolutionary materialism advocates will answer to it. END

PS: I am facing a security headache, so this post was completed on a Linux partition. Linux is looking better than ever, just now. as a main OS . . .

Comments
kairosfocus wrote: (I think that part of the problem is that those committed to darwinism or its relatives, find great difficulty understanding what design thinkers are saying, as they are filtering through darwinist glasses.) ------------------------------------------------------- One of the mind blowing comments made to me by a well known scientists,lecturer, on 'evolution' and the state of science today. Said to me, "we are not set up to detect ID, we don't need to to." That statement, set me back a bit, it is not about evidence, or what the science says, its about attitude, or should I say altitude. So discussing the science (evidence)will not get you anywhere. Its better to deal with the attitude. That they are the ones to tells us all the answers.And because they have done and learned a lot, they feel justified, that in time they will know it all. They have faith. Why else would some say 'evolution' is a fact. It is attitude. http://patternsofcreation.weebly.com/MrDunsapy
January 1, 2012
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Actually just about anything can be put into a nested hierarchy. It all depends on your defining criteria. That said common descent does not predict a nested hierarchy based on traits for the simple reason that a nested hierarchy based on traits requires the traits be immutable and additive and evolution is not like that.Joe
January 1, 2012
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I think the truth is that you do not understand evolution, Joe.
Great another false accusation.
Do you think that mammals evolve into non-mammals?
No. But if the ToE is true they may.
As for vestigial legs on whales, yes there are.
Wrong again. Evos only think they are legs because they think whales evolved from some land mammal that had legs. The structures could very well be from FLIPPERS that are no longer developing.
And yes, I know that antibiotic resistance doesn’t help me. It’s a great problem, but luckily evolutionists understand it, and so help combat it.
You are clueless. Using baraminology we can fight bacteria and anti-biotic resistance. The fossil record can't have genetic confirmation as no DNA is recovered with the fossils and no geneticist even knows if teh changes required are even possible. Yes take a beak and go out and gather some evidence to support your nonsensical claims.Joe
January 1, 2012
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Elizabeth, "Speciation" is ambiguous at best. Ya see we do have so-called seoarate species that can interbreed. And if speciation = macroevolution then YECs accept macroevolution because they accept speciation. However with speciation there has never been any observed new body plans with new body parts.Joe
January 1, 2012
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I think the truth is that you do not understand evolution, Joe. Do you think that mammals evolve into non-mammals? Or dogs into non-dogs? They don't. No-one claims they do. As for vestigial legs on whales, yes there are. Saying there aren't doesn't make it so. I've seen vestigial leg bones on a whale skeleton with my own eyes. And yes, I know that antibiotic resistance doesn't help me. It's a great problem, but luckily evolutionists understand it, and so help combat it. If it was left to authors like the one in your link, we'd be in much greater trouble. Pseudo-scientists who are prepared to sign a declaration that they will only support conclusions that conform to a priori assumptions. And the fossil record does have genetic confirmation - remarkably so. Genetic phylogenies have remarkable congruence with phenotypic phylogenies except in certain very well-accounted for instances. I'll take a break from responding to you, I think. It seems to be a waste of both my time and yours. Have a very happy new year. Cheers LizzieElizabeth Liddle
January 1, 2012
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Nested hierarchy of vehicles: Vehicles without an engine-> land (snow (skis), ice(skates), ground ->wheeled(cart), water(row boat)(sail boat)(kayak)(canoe), air (landing gear – snow(ski glider), ice(ice sailboat), water(float glider), ground( wheels)(glider)) Vehicles with engines-> land (snow(skidoo), ice, ground (car), water(boat), air-> landing gear- snow (skiplane), water (floatplane), ground ->wheels (aeroplane)
Superset = Vehicles (as defined by Elizabeth) next level has two sets- one for powered vehicles (engines) and one for no engines- (human powered). Under the engine set we have three sets for land vehicles, air vehicles and water vehicles. Under the no-engine set we have the same three sets. From there each vehicle sits nicely in its own set.Joe
January 1, 2012
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Elizabeth: Yes, you can observe incremental evolution – look at the Grants’ work in on the Galapagos finch beaks, and how mean size tracked mean seed size in every generation. And you can then investigate the alleles responsible for those morphological changes, as has been done. The problem is that you don't know what molecular changes are responsible for the size of the finch beaks, or what is the molecular difference between alleles. On aminoacid can make a big phenotypic difference, for instance by inactivationg a fundamental protein, and yet the change is very simple at molecular level. On the contrary, the emergence of a completely new protein is a very complex event, but the phenotipic effects need not be huge. That's why discussions about the phenotype are pointless, if we don't know the moleculare cause. It's about causes that we are discussing. (By the way, have you answered my epistemologic comments?)gpuccio
January 1, 2012
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What overlaps? Everyting is in its own set. You are clueless, thanks.Joe
January 1, 2012
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You seem to be using a non-standard definition of "speciation". Could you give it?Elizabeth Liddle
January 1, 2012
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Whaa....???!!!! Sheesh, Joe. Gimme a chance. No, you have not constructed a "nested hierarchy". Look at all the overlaps. Try again. Use an actual Venn diagram. Then draw a circle round vehicles with skis. That circle does not nest. Do the same for wheels. That circle does not nest. It seems that you don't understand the principle of nested hierarchies. I'll try to explain, but it'll have to wait.Elizabeth Liddle
January 1, 2012
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Elizabeth: But there are a couple of things that need clearing up anyway. If “speciation” is a “macroevolutionary event” that is a very different definition from an event requiring a > 150 bit step change in dFCSI (however you ascertain that). Please, read again what I wrote: "Speciation is usually considered a macroevolutionary event, and I would agree, but that’s exactly because there are clear, observable and quantifiable genomic differences between species." As you can see, I consider speciation a macroevolutionary event because, in general, it offers evidence of "observable and quantifiable genomic differences" that are in themselves macroevolutionary. For instance, the occurrence of even one new de novo protein in a species would most probably be a macroevolutionary event, if we can confirm that the protein has at leal 150 bits of dFSCI. We have at least 1000 macroevolutionary events observed after OOL (the emergence of each of the about 1000 new protein domains during natural history). Isn't that enough to begin?gpuccio
January 1, 2012
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Finches "evolving" into finches, guppies "evolving" into guppies and fruit flies "evolving" into fruit flies all support baraminology, not universal common descent. Also there aren't any vestigal legs on whales, anti-biotic resistance doesn't help you and the fossil record requires genetic confirmation which it currently lacks. But the real problem is that you do not understand ID nor Creation- neither reject evolution outright.Joe
January 1, 2012
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OK Elizabeth I will take your silence as an admission that one can construct a nested hierarchy out of vehicles. Anything else I can help you with?Joe
January 1, 2012
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There still isn't any genetic data that demonstrates one type of vision system can evolve into another type. That should be a big problem for your theory.Joe
January 1, 2012
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You are mistaken as the theory of evolution only expects a nested hierarchy based on the vague notion of "all life". Based on characteristics we would expect a Venn diagram.Joe
January 1, 2012
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Actually what you’re describing is a light-sensitive spot plus the nerves to transmit the message plus the behavior to respond. In how many iterations did the organism ignore the shadow or perhaps move toward it? In how many iterations did it do something totally unpredictable like flip on its back or spin in circles when it sensed a shadow? It sounds silly,
It's not silly at all. Just look at the way moths fly into candleflames. They haven't yet evolved to respond appropriately. They may. The selective pressure is there. But recall that sight must have been preceded by other sensory systems and cell-signalling systems.
but so many complex behaviors, in order to have evolved, must have begun with seemingly odd responses to stimuli. Take the bird that feigns injury to lure away predators. That must have begun with a behavior that would be far less beneficial, like simply fleeing, or feigning injury right on top of the nest.
Yes indeed.
Anything can be simple if you oversimplify it. It’s when we apply the scrutiny of critical thought which is so sorely lacking from this field and actually attempt to apply the theory that we see how far short it falls.
Yes, anything can be simple if you oversimplify it. But that doesn't mean that simple systems can't be precursors to complex systems, or that coevolution can't happen. The palaeontological record suggests exactly that.Elizabeth Liddle
January 1, 2012
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Right, but you asked what use half an eye was. I have told you that :) But you are absolutely right that any eye, even a fractional eye, is no use unless it is connected to the rest of the system. But that is a different question - you askes what use half an eye was. I hope that is now clear. Right, so let's look not at "half an eye" but a rudimentary visual system, which makes a lot more sense. The most elementary visual system is probably one in which a light sensitive spot reacts to light in such a way that a muscular movement is triggered in the organism. In other words the light-sensitive cells would need to have some way of signalling to muscle cells. Could be chemical, could be ionic. Both kinds of signalling systems were probably already in place before eyes first appeared, certainly chemical signalling systems, as they are essential for multi-cellular life. For any prey creature for whom a passing shadow tended to be caused by a predator, a creature who reflexively moved when a shadow fell might pass on more offspring. That's just a hypothetical. But it is fairly easy to set up simulations in which such visual-systems evolve. And once you have a functional connection between a light (or sound, or chemical) detector and the motor system you have the beginnings of a functional sensory system. And once you have a beneficial sensory system, it can evolve. I'll have to check the extant animals. I'm not a zoologist, so I can't remember them offhand. Jellyfish are one - I think they just have a light sensitive pigment in a depression. And I think a nautilus has a pinhole eye. There's a book, I think, called "Animal Eyes". Can't remember the author. I got it from the library a while back. Ah, here it is: http://ukcatalogue.oup.com/product/9780198575641.doElizabeth Liddle
January 1, 2012
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Dr. Liddle, The spot on the skin surface is not capable of telling its owner anything unless, as Scott Andrew pointed out, there’s a system in place by which it A) can convey its message and B) there’s reason enough for the owner to respond to it and does so properly. What was the purpose of that spot-message-conveying-system before the spot came along? And if the system wasn't in place what was the purpose of the spot before the message system was in place? Or if they developed simultaneously, how? Finally, what are those extant animals you mentioned?lpadron
January 1, 2012
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This is true. But you must know the genetic basis, or at least have a few solid examples, to attribute that evolution to a process of genetic variations and selection. You know neither, and yet attribute the changes to both. How? Why?
We have overwhelming evidence that "genes" - the units responsible for inheritance - are molecular. Are you disputing that heritable phenotypic features are genetic as we now understand the word (i.e. carried by DNA sequences)? Why should we doubt that incremental morphological changes observed in the fossil records are genetic? Do you, in fact, doubt it? Now, we have discovered a lot, recently, about just how genes combine to produce developmental processes, and also about epigenetic effects, which is fascinating, and complicates the picture somewhat, but that has only enhanced our understanding of the kinds of genetic variation, particularly to regulatory genes, that give rises to morphological variance. And we can also make genetic phylogenies that can tell us a lot about what genes are responsible for what features in extant animals, and when they must have appeared, doing what, in their forebears. But clearly we can't actually do genetics on fossils because there is no genetic material.
How can you in one sentence state that you can observe incremental evolution while conceding that you cannot observe the increments or even know that they are? That is contradictory and makes absolutely no sense.
Yes, you can observe incremental evolution - look at the Grants' work in on the Galapagos finch beaks, and how mean size tracked mean seed size in every generation. And you can then investigate the alleles responsible for those morphological changes, as has been done. What on earth is contradictory about it?Elizabeth Liddle
January 1, 2012
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OK, well, it is difficult to keep track of these multi-person discussions! But Petrushka's response seemed to me an appropriate one to Bruce's - your objection to his response brought up a quite different issue. But there are a couple of things that need clearing up anyway. If "speciation" is a "macroevolutionary event" that is a very different definition from an event requiring a > 150 bit step change in dFCSI (however you ascertain that). Speciation events often result in populations that are far more similar, at least at first, than, for example a descendent population and its ancestral population, and does not require any "step changes". Nor, IMO, does adaptation, but that is what is under dispute! So I'm still finding your argument circular: you are first of all saying: macroevolution is evolution that requires a step change, then challenging us to account for it, without first demonstrating that such step changes ever actually occurred! You certainly can't assume that speciation events require such step changes, and, as Petrushka points out, new features such as feathers also show incremental evolution, not step changes. If you want us to explain step changes, please present evidence show that they actually occurred!Elizabeth Liddle
January 1, 2012
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Elizabeth: No. You are wrong. Petrushka cited evidence that “advances” generally regarded as “macroevolutionary” by IDists (feathers; the mammalian ear) emerged incrementally i.e. by micro-evolutionary steps. As an IDist, I have never regarded a phenotypic "advance" whose molecular basis is not known as "macroevolutionary". I don't know if othyers have done that, but I can respond for myself. The macroevolutionary events I usually debate (as you should know) are the emergence of new basci protein domains. Speciation is usually considered a macroevolutionary event, and I would agree, but that's exactly because there are clear, observable and quantifiable genomic differences between species. The relation between genomic information and some morphologic aspects of phenotype is sittl not understood, so I maintain that we cannot use those examples to debate our causl explanatory models. I have always stated that, and I will go on stating that. Us: but our whole point is that there aren’t big step changes! Look at these incremental changes that amount to big changes in the long term! You don't know the cause of the incremental change. You don't know the cause of the big change. You cannot know, therefore, how incremental, or how big, those changes are at genomic level. Is that clear?gpuccio
January 1, 2012
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Easily. You don’t need to know the genetic basis of a phenotypic feature to observe incremental evolution over time.
This is true. But you must know the genetic basis, or at least have a few solid examples, to attribute that evolution to a process of genetic variations and selection. You know neither, and yet attribute the changes to both. How? Why? How can you in one sentence state that you can observe incremental evolution while conceding that you cannot observe the increments or even know that they are? That is contradictory and makes absolutely no sense.ScottAndrews2
January 1, 2012
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That’s only because your side is not really interested in explaining what is observed (functional information in biology at he molecular level), and is content with a non explanation. Your choice, but we beg to differ.
Not in the least, gpuccio! That is completely untrue. Evolutionary genetics is a huge field, as is evo devo! Let's recap: Bruce David: In the entire list, there is no actual evidence that RM/NS has ever produced a single macroevolutionary advance in living organisms. Petrushka: There is evidence that the mammalian inner ear bones evolved by incremental change. There is evidence that feathers evolved incrementally. There is evidence that the whales spout moved incrementally from the front of the face to the top of the head. gpuccio: Strange that in all the cases you present the molecular basis is not known. How can you talk of microevolution or macroevolution if you don’t know the molecuolar vatiation implied? Me: Easily. You don’t need to know the genetic basis of a phenotypic feature to observe incremental evolution over time. Nor do you need to know the genetic basis of a feature to infer that it is heritable. The word “gene” was coined long before its molecular basis was discovered. gpuccio: That’s exactly my point. If you don’t know the molecular basis, you don’t know the informational content of the variation. It can be incremental, it can be heritable, but we cannot classify it as microevolution or macroevolution. You will probably remember that I have given an operational distinction based on dFSCI. Me: But it’s your side, not ours, that is attempting to so classify! gpuccio: That’s only because your side is not really interested in explaining what is observed (functional information in biology at he molecular level), and is content with a non explanation. Your choice, but we beg to differ. In other words, as I read it, Bruce challenged evolutionists to come up with a "macroevolutionary advance" (not a term that evolutionists generally use). Petrushka cited evidence that "advances" generally regarded as "macroevolutionary" by IDists (feathers; the mammalian ear) emerged incrementally i.e. by micro-evolutionary steps. In other words, that microevolution, which your side generally regard as accountable by "RM+NS" is also responsible for macroevolutionary change. Then you object that we don't know the molecular basis for these changes. I agree, but say that that doesn't stop us observing incremental, i.e. microevolutionary steps. You then say that you can't tell the difference between macro and micro unless you know the genetics because genetics is how you define macro and micro evolution! See those goal posts move! So I say: but that's your definition. If you want to claim that some changes demand big step changes in dFCSI, feel free, but that's your case, not ours. Our case is simply that macroevolutionary change occurs in microevolutionary steps, which are perfectly explicable, as we all agree, by "RM+NS". Or, as I would much prefer to describe it (because much more direct): heritable variance in reproductive success within the current environment. You guys agree that happens on an observable micro scale. All we are doing is showing that if you extend that micro scale longitudinally, you end up with "macro" evolution - as long as you don't move those goal posts! Obviously WE can't demonstrate that evolutionary processes are capable of step changes of 150 bits because we don't think it is! So the whole thing amounts to: You guys: show how evolution can produce macroevolution. Us: look at incremental evolution of feathers. You guys: no, that's not macro evolution, macro evolution is when you get a step increas of 150 bits. Us: but our whole point is that there aren't big step changes! Look at these incremental changes that amount to big changes in the long term! You guys: you aren't interested in genetics. Us: ???????????!!!!!!!!!Elizabeth Liddle
January 1, 2012
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A light sensitive spot on the skin surface is capable of telling its owner when a shadow passes over – potential warning of a predator, or, conversely, information that the owner has reached shelter.
Actually what you're describing is a light-sensitive spot plus the nerves to transmit the message plus the behavior to respond. In how many iterations did the organism ignore the shadow or perhaps move toward it? In how many iterations did it do something totally unpredictable like flip on its back or spin in circles when it sensed a shadow? It sounds silly, but so many complex behaviors, in order to have evolved, must have begun with seemingly odd responses to stimuli. Take the bird that feigns injury to lure away predators. That must have begun with a behavior that would be far less beneficial, like simply fleeing, or feigning injury right on top of the nest. Anything can be simple if you oversimplify it. It's when we apply the scrutiny of critical thought which is so sorely lacking from this field and actually attempt to apply the theory that we see how far short it falls.ScottAndrews2
January 1, 2012
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Elizabeth: That's only because your side is not really interested in explaining what is observed (functional information in biology at he molecular level), and is content with a non explanation. Your choice, but we beg to differ.gpuccio
January 1, 2012
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A light sensitive spot on the skin surface is capable of telling its owner when a shadow passes over - potential warning of a predator, or, conversely, information that the owner has reached shelter. A light sensitive spot in a slight depression is capable of relaying directional information about the source of the shadow (and may be the reason our brains are wired so strangely, with the left hemispher controlling the right side of our bodies and vice versa). A light sensitive spot in a deeper depression is eve nmore capable of relaying directional information. A light sensitive spot in a depression with only a pinhole is capable of resolving an actual image. A light sensitive spot covered with translucent tissue is better protected than one without. A light sensitive spot in which the translucent tissue is concave will focus the light more sharply, improving resolution. A light sensitive spot on a body part that can be angled independently is capable of being focusssed on a particular region of space, enabling the creature to maintain "fixation" while it moves towards the fixated point e.g. prey. Will that do for starters? There are extant animals with eyes like each of these.Elizabeth Liddle
January 1, 2012
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That’s exactly my point. If you don’t know the molecular basis, you don’t know the informational content of the variation. It can be incremental, it can be heritable, but we cannot classify it as microevolution or macroevolution. You will probably remember that I have given an operational distinction based on dFSCI.
But it's your side, not ours, that is attempting to so classify! As far as evolutionary theory goes, adaptation over short term and long term are essentially the same, except that over the long term, population-level selection starts to play a role, and speciation - the subdivision of populations into two non-interbreeding and thus independently adapting populations - also becomes apparent. It's just different scale views of the same phenomena. If you want to claim that microevolution and macroevolution are two different things based on some measure of informational content of - the step size? I'm not sure - then yes, of course, you will have to measure that. But that's your claim, not ours. All we are doing is showing that phenotypic features, such as the mammalian ear, evolved incrementally over many generations, just as finch beaks evolve incrementally to cope with new seed sizes, from generation to generation.Elizabeth Liddle
January 1, 2012
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Elizabeth: That's exactly my point. If you don't know the molecular basis, you don't know the informational content of the variation. It can be incremental, it can be heritable, but we cannot classify it as microevolution or macroevolution. You will probably remember that I have given an operational distinction based on dFSCI.gpuccio
January 1, 2012
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Easily. You don't need to know the genetic basis of a phenotypic feature to observe incremental evolution over time. Nor do you need to know the genetic basis of a feature to infer that it is heritable. The word "gene" was coined long before its molecular basis was discovered. And, in fact, we know very little about the genetic basis for any phenotypic feature. What we do know now is a fair bit about which genes, and alleles of which genes, are implicated in the development of phenotypic features - it's what evo-devo is all about, but it's still very much in its infancy. Phenotypic features are the result of highly complex interactions between many genes and the expressed products of many others.Elizabeth Liddle
January 1, 2012
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Petrushka: Strange that in all the cases you present the molecular basis is not known. How can you talk of microevolution or macroevolution if you don't know the molecuolar vatiation implied?gpuccio
January 1, 2012
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